| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6002128 | Thrombosis Research | 2013 | 7 Pages |
Abstract
Brain death causes hypercoagulation; however, inhibition of TF does not reverse the coagulopathy. Thus, TF release does not seem to be the primary cause of this hypercoagulation. Minor changes in the levels of protein C suggest that the protein C pathway may be linked to the observed coagulopathy.
Keywords
ASISbrain deadCTIPCTCCPPAI-1ETPICPAPTTMCFBSAROTEMbovine serum albuminstandard deviationmaximum velocityMaximum clot firmnessTimeactivated partial thromboplastin timeClotting timeprothrombin timeintracranial pressuremean arterial pressureCerebral perfusion pressurecorn trypsin inhibitorPlasminogen activator inhibitor-1mapendogenous thrombin potential
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Authors
Christine L. Hvas, Christian Fenger-Eriksen, Søren Høyer, Benny Sørensen, Else Tønnesen,