Article ID Journal Published Year Pages File Type
6018324 Experimental Neurology 2013 9 Pages PDF
Abstract
► Infection drastically alters pathogenic mechanism of hypoxic-ischemic brain injury. ► Intranasal delivery of anti NFkB peptides blocks infection sensitized HI injury. ► Results suggest a safe, brain targeted anti neuroinflammatory therapy.
Keywords
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Life Sciences Neuroscience Neurology
Preview
Intranasal delivery of cell-penetrating anti-NF-κB peptides (Tat-NBD) alleviates infection-sensitized hypoxic-ischemic brain injury
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