| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6019305 | Experimental Neurology | 2010 | 5 Pages |
Abstract
This result points at secretion as the main mechanism of cyst filling, and NKCC1 as the key candidate of fluid transport. Based on these findings, we hypothesize that selective NKCC1 inhibitors could be used in preventing expansion of temporal AC.
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Authors
Christian A. Helland, Mads Aarhus, Per Knappskog, Lisa K. Olsson, Morten Lund-Johansen, Mahmood Amiry-Moghaddam, Knut Wester,