Article ID Journal Published Year Pages File Type
6022833 Neurobiology of Disease 2011 11 Pages PDF
Abstract
►Increased Ca2+ in mitochondria, ER and cytoplasm preceded SOD1G93A inclusions. ►Increased cytosolic Ca2+ promoted SOD1G93A inclusion formation. ►Proteasome inhibition caused mitochondrial abnormalities, but independent of Ca2+. ►Geldanamycin prevented inclusions and mitochondrial rounding, but not altered Ca2+.
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