| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6037368 | NeuroImage | 2010 | 8 Pages |
Abstract
Deficiency of FXII allows neocortical reperfusion after tMCAO and rescues brain tissue by this mechanism. This study supports the concept of FXII as a promising new target for stroke prevention and therapy.
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Authors
M. Pham, C. Kleinschnitz, X. Helluy, A.J. Bartsch, M. Austinat, V.C. Behr, T. Renné, B. Nieswandt, G. Stoll, M. Bendszus,