| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6051093 | Archives of Oral Biology | 2013 | 7 Pages |
Abstract
From this result, PPARγ acts to inhibit bone destruction in AH26-induced bone cells. Therefore, the anti-inflammatory and anti-osteoclastic character of PPARγ might be applicable for healing periapical lesions more rapidly or reducing the induction of cellular inflammation caused by some endodontic sealers.
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Authors
Tae-Gun Kim, Young-Hee Lee, Govinda Bhattari, Nan-Hee Lee, Kwang-Won Lee, Ho-Keun Yi, Mi-Kyung Yu,