| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6051570 | Archives of Oral Biology | 2011 | 8 Pages |
Abstract
These data support a model wherein ZA treatment impedes oral wound healing by blocking the growth and migratory capacity of oral fibroblasts as well as downregulating the transcription of type-I collagen, functions necessary to deposit the granulation tissue needed for re-epithelization. Therefore, BP released from bone following tooth extraction may delay wound healing of the oral mucosal barrier and contribute to BPONJ pathogenesis.
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Authors
Matthew J. Ravosa, Jie Ning, Yueying Liu, M. Sharon Stack,