| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6052035 | Archives of Oral Biology | 2012 | 10 Pages |
Abstract
These findings suggest a role for COX-2 and IL-10 in the etiopathogenesis of oral PG and indicate that LCH and non-LCH histological subtypes represent different stages in the evolution of a single lesion with varying degrees of proliferative, angiogenic, and inflammatory activity.
Keywords
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Authors
Diana M. Isaza-Guzmán, Clara B. Teller-Carrero, MarÃa P. Laberry-Bermúdez, Leonor V. González-Pérez, Sergio I. Tobón-Arroyave,