| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6092903 | Gastroenterology | 2016 | 36 Pages |
Abstract
Enteric glial cells from patients with CD have reduced production of 15-HETE, which controls IEB permeability by inhibiting adenosine monophosphateâactivated protein kinase and increasing expression of zonula occludens-1.
Keywords
PBSDMEMAMPKROG15-HETEEnteric glial cellERKZO-1TEERIBDPGE2EGCDulbecco’s modified Eagle medium15-hydroxyeicosatetraenoic acidArachidonic acidPolyunsaturated fatty acidPUFALOXCrohn’s diseaseInflammatory bowel diseaseintestinal epithelial cellenteric nervous systemlipoxygenaseIntestinal Epithelial BarrierPhosphate-buffered salinetransepithelial electrical resistanceZonula occludens-1Prostaglandin E2IECUlcerative colitisextracellular signal-regulated kinase
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Authors
Camille Pochard, Sabrina Coquenlorge, Julie Jaulin, Nicolas Cenac, Nathalie Vergnolle, Guillaume Meurette, Marie Freyssinet, Michel Neunlist, Malvyne Rolli-Derkinderen,