| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6093361 | Gastroenterology | 2015 | 14 Pages |
Abstract
Increased production of NOS2 in enteric glia contributes to the dysregulation of intestinal ion transport in mice with colitis. Blocking enteric glial function in these mice restores epithelial barrier function and reduces bacterial translocation.
Keywords
SMTCNOS1NOS2TNBSENSTTXEFsNOSIBDVIPISCDSSS-methyl-l-thiocitrulline1400WcAMPCyclic adenosine monophosphateTrinitrobenzene sulfonic acidMyenteric plexusinflammationInflammatory bowel diseasetetrodotoxinElectrical field stimulationEnteric glial cellsinducible nitric oxide synthaseneuronal nitric oxide synthasedextran sodium sulfateenteric nervous systemShort-circuit currentNitric oxidenitric oxide synthasevasoactive intestinal peptide
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Authors
Sarah J. MacEachern, Bhavik A. Patel, Catherine M. Keenan, Michael Dicay, Kevin Chapman, Donna-Marie McCafferty, Tor C. Savidge, Paul L. Beck, Wallace K. MacNaughton, Keith A. Sharkey,