| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6094166 | Gastroenterology | 2015 | 14 Pages |
Abstract
Based on studies of Foxl1-Cre;RosaYFP/iDTR mice, Foxl1+ HPCs and/or their descendants are required for the development of cholangiocytes and hepatocytes in liver after CDE diet-induced injury.
Keywords
4-HNEcholine-deficient, ethionine-supplementedHNF4αCK19γH2AXYFPALTCDEGGTHPC4-hydroxy-2-nonenalddCHPCsγ-glutamyltransferaseASTAspartate aminotransferaseAlanine aminotransferaseALPAlkaline phosphataseCreERterminal deoxynucleotidyl transferase–mediated deoxyuridine triphosphate nick-end labelingNonalcoholic fatty liver diseaseTUNELOval cellshepatic progenitor cellcytokeratin 19hepatocyte nuclear factor 4 alphaMouse modelyellow fluorescent protein
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Authors
Soona Shin, Naman Upadhyay, Linda E. Greenbaum, Klaus H. Kaestner,