| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6096742 | Gastroenterology | 2011 | 9 Pages |
Abstract
These studies identify defective expression and function of CD116 as a distinguishing feature of IBD and implicate an associated defect in innate immune responses toward granulocyte-macrophage colony-stimulating factor.
Keywords
TNFMFIROCM-CSFpSTAT3GM-CSFRheumatoid arthritisinflammationrecombinant humaninterleukinIntestinegranulocyte-macrophage colony-stimulating factorgranulocyte colony-stimulating factorGrowth factorG-CSFtumor necrosis factorLeukocytemacrophage colony-stimulating factormean fluorescence intensitypolymerase chain reactionPCRreceiver operator characteristic
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Authors
Jonathan I. Goldstein, Douglas J. Kominsky, Nicole Jacobson, Brittelle Bowers, Kirsten Regalia, Gregory L. Austin, Melinda Yousefi, Michael T. Falta, Andrew P. Fontenot, Mark E. Gerich, Lucy Golden-Mason, Sean P. Colgan,