| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6109607 | Journal of Hepatology | 2009 | 7 Pages |
Abstract
The effects of BDL apparently result in part from down-regulation of A2A adenosine receptor. CBD reverses these effects through activation of this receptor, leading to compensation of the ligation effect.
Keywords
i.p.arachidonoyl ethanolamide2-arachidonoylglycerolBDL2-AGBSFCBDCBRLPSTAABDNFECsanandamideinflammationendocannabinoidsGene expressionIntraperitoneallyCognitionBrain-derived neurotrophic factoractivitylipopolysaccharidebile duct ligationthreshold cycleCannabidiolAdenosine receptorscannabinoid receptors
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Authors
Iddo Magen, Yosefa Avraham, Zvi Ackerman, Lia Vorobiev, Raphael Mechoulam, Elliot M. Berry,