| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6109677 | Journal of Hepatology | 2009 | 12 Pages |
Abstract
The lower level of cytokines/chemokines in Gas6â/â mice after CCl4 injury, is the consequence of an inhibitory signal arising from Axl receptor overexpression, leading to delayed liver repair in deficient mice.
Keywords
PCNAASMAIL-6VCAM-1CCR2MCP-1CCl4HSCalpha smooth muscle actinProliferating Cell Nuclear AntigeninflammationAxlinterleukin 6Liver repairtumor necrosis factor alphaHepatic stellate cellKupffer cellsTNF-αvascular cell adhesion molecule-1wild-typemonocyte chemoattractant protein 1Growth arrest-specific protein 6Carbon tetrachlorideGas6
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Authors
Fouad Lafdil, Marie-Noële Chobert, Vanessa Deveaux, Elie-Serge Zafrani, Philippe Mavier, Toru Nakano, Yannick Laperche, Arthur Brouillet,