| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6125037 | Osteoarthritis and Cartilage | 2014 | 10 Pages |
Abstract
This study supports the implication of BSP in the pathology of OA and suggests that it could be a key mediator of the hypertrophic chondrocytes-induced angiogenesis. To control chondrocyte hypertrophic differentiation is promising in the treatment of OA.
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Authors
L. Pesesse, C. Sanchez, D.A. Walsh, J.-P. Delcour, C. Baudouin, P. Msika, Y. Henrotin,