| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6241482 | Respiratory Medicine | 2015 | 8 Pages |
Abstract
In SRA IL-18 is possibly involved in chronic airway inflammation through an eosinophil independent pathway. The decreased levels of IL-18 in SRA support the hypothesis of deregulated inflammasome activation, justifying the susceptibility of these patients for bacterial colonization or infection.
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Authors
Nikoletta Rovina, Efrossini Dima, Petros Bakakos, Eleni Tseliou, Konstantina Kontogianni, Spyros Papiris, Antonia Koutsoukou, Nikolaos G. Koulouris, Stelios Loukides,