| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6249154 | Transplantation Proceedings | 2011 | 6 Pages |
Abstract
MIL maintained renal tissue blood flow by its vasodilatory effect, suppressed expression of TNF-α mRNA by increasing intracellular cyclic adenosine monophosphate, and ultimately decreased tubular cell apoptosis, thus protecting renal function after warm I-R injury.
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Authors
T. Nishiki, H. Kitada, Y. Okabe, Y. Miura, K. Kurihara, S. Kawanami, M. Tanaka,