| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6265323 | Brain Research | 2011 | 10 Pages |
Abstract
âºInhibition of NO synthases enhances TRH mRNA levels in hypothalamic PVN. âºInhibition of NO synthases reduces serum thyrotropin concentration. âºThe effect of NO may include a direct action on hypothalamic neurons. âºInhibition of NO synthase blocked cold stress-induced increase in TSH release. âºProduction of NO may control the activity of the HPT axis.
Keywords
AVPhypothalamus–pituitary–thyroidACTHNILNG-nitro-l-arginine methyl esterMBHTSHhptNPYNOSl-NAMECold stressoxytocintrhthyrotropinPVNHPAsodium nitroprussideNitric oxidenitric oxide synthaseparaventricular nucleus of the hypothalamusparaventricular nucleusThyrotropin releasing hormoneHypothalamusMediobasal hypothalamushypothalamus–pituitary–adrenalAnterior pituitaryarginine vasopressinSNPcorticotrophinNeuropeptide Y
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Authors
Rosa Maria Uribe, Miguel Cisneros, Miguel Angel Vargas, Leticia Lezama, Antonieta Cote-Vélez, Patricia Joseph-Bravo, Jean-Louis Charli,