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The role of TWEAK/Fn14 signaling in the MPTP-model of Parkinson's disease

Article ID Journal Published Year Pages File Type
6271366 Neuroscience 2016 7 Pages PDF
Abstract
These findings suggest that TWEAK signaling might be an aspect of MPTP-mediated neuropathology and be involved in the overall neurodegenerative pathology of PD.
Keywords
DOPACMPTP3,4-dihydrophenylacetic acidNF-κBPBSfibroblast growth factor-inducible 14Fn14TNFR1TWEAKTBSHRPCOX-21-methyl-4-phenyl-1,2,3,6-tetrahydropyridinePCASNpcPerchloric acidParkinson’s diseaseTris-buffered salinesubstantia nigra pars compactatumor necrosis factor-alphatyrosine hydroxylasestandard error of the meanTNF-αnuclear factor-kappaBPhosphate-buffered salineSEMHorseradish peroxidasehigh-performance liquid chromatographyHPLCTNF receptor 1
Related Topics
Life Sciences Neuroscience Neuroscience (General)
Preview
The role of TWEAK/Fn14 signaling in the MPTP-model of Parkinson's disease
Authors
S. Mustafa, H.L. Martin, L. Burkly, A. Costa, M.L. Martins, M. Schwaninger, P. Teismann,
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Journal
Neuroscience
Journal: Neuroscience
Related Categories
DOPAC
MPTP
3,4-dihydrophenylacetic acid
NF-κB
PBS
fibroblast growth factor-inducible 14
Fn14
TNFR1
TWEAK
TBS
HRP
COX-2
1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine
PCA
SNpc
Perchloric acid
Parkinson’s disease
Tris-buffered saline
substantia nigra pars compacta
tumor necrosis factor-alpha
tyrosine hydroxylase
standard error of the mean
TNF-α
nuclear factor-kappaB
Phosphate-buffered saline
SEM
Horseradish peroxidase
high-performance liquid chromatography
HPLC
TNF receptor 1
Behavioral Neuroscience
Biological Psychiatry
Cellular and Molecular Neuroscience
Cognitive Neuroscience
Developmental Neuroscience
Endocrine and Autonomic Systems
Neurology
Neuroscience (General)
Sensory Systems
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