| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6272500 | Neuroscience | 2015 | 16 Pages |
Abstract
Thus, striatal TH+ cells and serotonergic terminals may cooperatively synthesize DA and subsequently contribute to supraphysiological synaptic DA concentrations, an accepted cause in LID pathogenesis.
Keywords
AADCaccumbensMFBNHSΔFosB6-HydroxydopamineLIDaromatic acid decarboxylaseTH+6-OHDAl-3,4-dihydroxyphenylalaninel-DOPASNpcStriatumAIMSmedial forebrain bundleParkinson’s diseasesubstantia nigra pars compactatyrosine hydroxylaseabnormal involuntary movementsabnormal involuntary movementDopamineL-DOPA-induced dyskinesianormal horse serumoptical density
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Authors
U. Keber, M. Klietz, T. Carlsson, W.H. Oertel, E. Weihe, M.K.-H. Schäfer, G.U. Höglinger, C. Depboylu,