| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6273000 | Neuroscience | 2015 | 15 Pages |
Abstract
Correlating the results with existing literature on the metabolic functions of Schwann cells and axons generates testable hypotheses for the mechanism of hyperglycemic damage during anoxia and lead to discussions of the role for a metabolic shuttle between Schwann cells and axons as well as a potential important role of glycogen.
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Neuroscience
Neuroscience (General)
Authors
M.M. Stecker, M.R. Stevenson,