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Chronic treatment with lithium does not improve neuromuscular phenotype in a mouse model of severe spinal muscular atrophy

Article ID Journal Published Year Pages File Type
6274499 Neuroscience 2013 61 Pages PDF
Keywords
SOD1SMNCGRPNMDAGSK-3βN-methyl-d-aspartateLiClmotoneuronGFAPTBSTVGLUT1PBSPI3KIBA1LithiumTibialis Anteriorspinal muscular atrophyamyotrophic lateral sclerosisphosphate bufferALSanalysis of varianceANOVAterminal deoxynucleotidyl transferase dUTP nick-end labelingfull-lengthTUNELvesicular glutamate transporter 1SMARoom temperaturesuperoxide dismutase 1Spinal cordSkeletal musclePhosphatidylinositol 3-kinaselithium chloridePhosphate-buffered salineionized calcium-binding adaptor molecule 1survival motor neuronGlial fibrillary acidic proteincalcitonin gene-related peptideGlycogen synthase kinase-3β
Related Topics
Life Sciences Neuroscience Neuroscience (General)
Preview
Chronic treatment with lithium does not improve neuromuscular phenotype in a mouse model of severe spinal muscular atrophy
Authors
E. Dachs, L. Piedrafita, M. Hereu, J.E. Esquerda, J. Calderó,
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Journal
Neuroscience
Journal: Neuroscience
Related Categories
SOD1
SMN
CGRP
NMDA
GSK-3β
N-methyl-d-aspartate
LiCl
motoneuron
GFAP
TBST
VGLUT1
PBS
PI3K
IBA1
Lithium
Tibialis Anterior
spinal muscular atrophy
amyotrophic lateral sclerosis
phosphate buffer
ALS
analysis of variance
ANOVA
terminal deoxynucleotidyl transferase dUTP nick-end labeling
full-length
TUNEL
vesicular glutamate transporter 1
SMA
Room temperature
superoxide dismutase 1
Spinal cord
Skeletal muscle
Phosphatidylinositol 3-kinase
lithium chloride
Phosphate-buffered saline
ionized calcium-binding adaptor molecule 1
survival motor neuron
Glial fibrillary acidic protein
calcitonin gene-related peptide
Glycogen synthase kinase-3β
Behavioral Neuroscience
Biological Psychiatry
Cellular and Molecular Neuroscience
Cognitive Neuroscience
Developmental Neuroscience
Endocrine and Autonomic Systems
Neurology
Neuroscience (General)
Sensory Systems
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