| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6274921 | Neuroscience | 2013 | 12 Pages |
Abstract
⺠Pathophysiological Abeta concentrations evoke anti-apoptotic effects. ⺠AChE-R mediates anti-apoptotic effects via mitochondrial hyperactivity. ⺠Toxic Abeta concentrations suppress AChE-R expression. ⺠Cholinergic dynamics (HACU and ChAT adaptation) support AChE-R mediated effects.
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Authors
G. Li, J. Klein, M. Zimmermann,