| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6275573 | Neuroscience | 2012 | 17 Pages |
Abstract
⺠Glial, neuronal and vascular lesions were investigated after status epilepticus. ⺠We found widespread loss of GFAP and NeuN, and increased laminin immunoreactivity. ⺠Astrocytic lesions and increased laminin immunoreactivity were significantly related. ⺠Neuronal lesions were not related to increased laminin signal. ⺠Vascular lesions affect more astrocytes than neurons.
Keywords
PBSPRHaccumbensPAMPCA3TLEET-1NeuNECoGSNRENTSPFCPUAOPCA1AMGGFAPPVAAcbMPORPccornu ammonis 3cornu Ammonis 1Amygdalareference electrodeelectrocorticographyendothelin-1Ischemiastatus epilepticusPirEpilepsystria terminalisSUBBBBBlood–brain barrierlateral septummedial septumdentate gyrustemporal lobe epilepsyEntorhinal cortexperirhinal cortexpiriform cortexphosphate buffer solutionREFVentral subiculumTenia tectazona incertacentral medial thalamic nucleusreuniens thalamic nucleussubparafascicular thalamic nucleusParaventricular thalamic nucleusPosterior hypothalamic nucleusmedial dorsal nucleusmedial preoptic nucleusanteroventral thalamic nucleusHippocampusCaudate putamenGlial fibrillary acidic proteinneuron-specific nuclear proteinPilocarpineGENKainateTegmentumGlobus pallidus
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Authors
F. Gualtieri, G. Curia, C. Marinelli, G. Biagini,