| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 6284018 | Neuroscience Letters | 2012 | 6 Pages |
Abstract
⺠Aberrant splicing of zebrafish psen1 leads to incorporation of intron sequence into transcripts. ⺠Zebrafish maintain normal psen1 transcripts and accumulate aberrantly spliced transcripts. ⺠Forcing Psen1 protein in zebrafish suppresses psen1 transcription. ⺠Maintenance of normal psen1 transcript levels appears important for Psen1 protein regulation.
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Authors
Morgan Newman, Svanhild Nornes, Ralph N. Martins, Michael T. Lardelli,