Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
7281636 | Brain, Behavior, and Immunity | 2014 | 9 Pages |
Abstract
The loss-of-function results support the hypothesis that endogenous IL-18 suppresses appetite and promote energy expenditure and lipid fuel substrate utilization not only during sickness, but also in healthy adults consuming high-fat diets.
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Authors
Eric P. Zorrilla, Bruno Conti,