Article ID Journal Published Year Pages File Type
7281636 Brain, Behavior, and Immunity 2014 9 Pages PDF
Abstract
The loss-of-function results support the hypothesis that endogenous IL-18 suppresses appetite and promote energy expenditure and lipid fuel substrate utilization not only during sickness, but also in healthy adults consuming high-fat diets.
Related Topics
Life Sciences Immunology and Microbiology Immunology
Authors
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