Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
7506977 | Drug and Alcohol Dependence | 2014 | 5 Pages |
Abstract
Age related deficits in electrophysiological components such as P20 or event-related gamma may be due to an interruption of normal neural maturation. Reduction of NMDAR signaling during adolescence leads to delayed-onset disruption of gamma oscillations and the P20 component of the ERP. Further, delayed onset of impairment following adolescent ketamine abuse suggests that methods could be developed to detect and treat the early effects of drug exposure prior to the onset of disability.
Related Topics
Life Sciences
Neuroscience
Behavioral Neuroscience
Authors
R.E. Featherstone, L.R. Nagy, C.G. Hahn, S.J. Siegel,