| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 7858172 | Dental Materials | 2018 | 16 Pages |
Abstract
TEGDMA inhibits CI in the respiratory chain, which explains effects induced by TEGDMA on redox homeostasis, apoptotic and necrotic cell deaths described in previous studies. Identification of the molecular target of TEGDMA may influence the development of relevant biomaterials and may induce new therapeutic strategies to control the adverse effects of resin monomers.
Keywords
α-KGDHSuccSLPSubstrate-level phosphorylationAP5ΔΨmCATα-KGFCCPDTNBOXPHOSANT5,5′-dithiobis(2-nitrobenzoic acid)BSADMSOHydrogen peroxideAdenylate kinasebovine serum albuminalpha-ketoglutarate dehydrogenasealpha-ketoglutarateOxidative stressRespirationATP productiondimethyl-sulfoxideCytotoxicitySuccinateOxidative phosphorylationrespiratory complex IMitochondrial membrane potentialCarboxyatractylateReactive oxygen species
Related Topics
Physical Sciences and Engineering
Materials Science
Biomaterials
Authors
Krisztina Mikulás, Péter Hermann, István Gera, Timea Komlódi, GergÅ Horváth, Attila Ambrus, László Tretter,