| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8215509 | International Journal of Radiation Oncology*Biology*Physics | 2016 | 25 Pages |
Abstract
These data demonstrate that miR-203 potentially controls DNA damage repair via the PI3K/AKT and JAK/STAT3 pathways and may collectively contribute to the modulation of radiation sensitivity in MGÂ cells by inhibiting DNA damage repair, prosurvival signaling, and epithelium-mesenchyme transition. Taken together, these findings demonstrate that miR-203 could be a target for overcoming the radiation resistance of GBM.
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Authors
Ji Hyun MD, Yeo Hyun PhD, David J. Lee, Dan Hyo MS, Ji Min Park, Hong-Gyun MD, PhD, In Ah MD, PhD,