| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8220214 | International Journal of Radiation Oncology*Biology*Physics | 2014 | 7 Pages |
Abstract
Both gene expression profiling and DNA DSB repair kinetics data imply that less-efficient repair of radiation-induced DSBs may contribute to the development of late normal tissue damage. Induction levels of DSB repair genes (eg, RAD51) may potentially be used to assess the risk for late radiation toxicity.
Related Topics
Physical Sciences and Engineering
Physics and Astronomy
Radiation
Authors
Bregje MSc, Suzanne E. MSc, Perry D. PhD, Jan Paul PhD, Lukas J.A. MD, PhD, Harry PhD, Nicolaas A.P. PhD,