Article ID Journal Published Year Pages File Type
8229022 International Journal of Radiation Oncology*Biology*Physics 2011 9 Pages PDF
Abstract
These data indicate that radioresistance of K-RAS mutated tumor cells is at least in part due to constitutive production of EGFR ligands, which mediate enhanced repair of DNA double-strand breaks through the EGFR-phosphatidylinositol-3-kinase-Akt cascade.
Related Topics
Physical Sciences and Engineering Physics and Astronomy Radiation
Authors
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