Nesfatin-1 functions as a switch for phenotype transformation and proliferation of VSMCs in hypertensive vascular remodeling

Article ID	Journal	Published Year	Pages	File Type
8258438	Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease	2018	39 Pages	PDF

Keywords

α-SMA PCNA nucleobindin 2 NUCB2 Ox-LDL CDK2 OPN N-acetyl-l-cysteine DAPI BCA HRP mTOR GAPDH NAC DBP PI3K SBP SDS CCK-8 DMEM FBS Janus kinase 4′,6-diamidino-2-phenylindole 5-ethynyl-2′-deoxyuridine EdU Dulbecco's modified Eagle's medium Proliferating Cell Nuclear Antigen Osteopontin α-smooth muscle actin bicinchoninic acid ELISA Enzyme-linked immunosorbent assay sodium dodecyl sulfate fetal bovine serum Shr cell counting kit-8 diastolic blood pressure systolic blood pressure mean arterial pressure Oxidized low-density lipoprotein Spontaneously hypertensive rats map mammalian target of rapamycin DHE Horseradish peroxidase Propidium iodide JAK cyclin dependent kinase 2 Glyceraldehyde phosphate dehydrogenase

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Nesfatin-1 functions as a switch for phenotype transformation and proliferation of VSMCs in hypertensive vascular remodeling

Authors

Qing-Bo Lu, Hui-Ping Wang, Zi-Han Tang, Han Cheng, Qiong Du, Yuan-Ben Wang, Wu-Bing Feng, Ke-Xue Li, Wei-Wei Cai, Li-Ying Qiu, Hai-Jian Sun,