| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8260256 | Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease | 2014 | 8 Pages |
Abstract
These results suggest that dysregulation of TGFBI is involved in the oncogenic activity of the sW182* mutant of the hepatitis B virus S gene.
Keywords
Dnmtcyclic AMP-responsive element binding proteinTGFBI5-AzacytidineHepatocarcinogenesisHBcAgCyclin D1GSEACOBRAGFPqPCRCREB5-AzaDNA methyltransferaseHCCGene Set Enrichment Analysiscombined bisulfite restriction analysisNonsense mutationhepatitis B core antigenHBVquantitative polymerase chain reactionhepatitis B virusgreen fluorescent proteinHepatocellular carcinoma
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Authors
Shih Sheng Jiang, Shiu-Feng Huang, Min-Syuan Huang, Yng-Tay Chen, Hsiang-Ju Jhong, Il-Chi Chang, Ya-Ting Chen, Jer-Wei Chang, Wen-Ling Chen, Wei-Chen Lee, Miin-Fu Chen, Chau-Ting Yeh, Isao Matsuura,