| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8261132 | Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease | 2013 | 10 Pages |
Abstract
⺠MiR-350 over-expressed through p38 and JNK protein synthesis. ⺠MiR-350 led to an increase in unphosphorylated NFATc3. ⺠Antagomir-350 could lower the levels of miR-350. ⺠Antagomir-350 could reverse the expression of target proteins. ⺠MiR-350 is a critical regulator of pathological cardiac hypertrophy and apoptosis.
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Authors
Yuzhi Ge, Shujuan Pan, Di Guan, Hong Yin, Yong Fan, Jingjing Liu, Shuhua Zhang, Hongjie Zhang, Lili Feng, Yunxia Wang, Ruxiang Xu, James Q. Yin,