Activation of classical estrogen receptor subtypes reduces tight junction disruption of brain endothelial cells under ischemia/reperfusion injury

Article ID	Journal	Published Year	Pages	File Type
8268096	Free Radical Biology and Medicine	2016	30 Pages	PDF

Keywords

H2DCF-DA YC-1 ZO-1 TEER DPN HIF-1α MCAO ERS FITC PPT 2′,7′-dichlorodihydrofluorescein diacetate 3-[4,5-dimethylthiazol-2-yl]-2,5-diphenyl tetrazolium bromide DMSO ERK1/2 extracellular signal-regulated kinase1/2 MTT OGD/R Small interfering RNA ROS siRNA TJs Ischemic reperfusion injury Tight junction tight junctions middle cerebral artery occlusion Dimethyl sulfoxide Blood–brain barrier BBB Endothelial cell hypoxia-inducible factor 1α Vascular endothelial growth factor Vascular Endothelial Growth Factor (VEGF)fluorescein isothiocyanate lactate dehydrogenase LDH transendothelial electrical resistance Paracellular permeability Zonula occludens-1 optical density Reactive oxygen species Estrogen receptor estrogen receptors

Related Topics

Life Sciences Biochemistry, Genetics and Molecular Biology Ageing

Activation of classical estrogen receptor subtypes reduces tight junction disruption of brain endothelial cells under ischemia/reperfusion injury

Authors

Jin A. Shin, Joo Chun Yoon, Minsuk Kim, Eun-Mi Park,