Left ventricular diastolic dysfunction in Nrf2 knock out mice is associated with cardiac hypertrophy, decreased expression of SERCA2a, and preserved endothelial function

Article ID	Journal	Published Year	Pages	File Type
8268873	Free Radical Biology and Medicine	2015	12 Pages	PDF

Keywords

eNOS SERCA2a IVRT PWV Nrf2 FMD Sarcoplasmic reticulum Ca2+-ATPase Flow-mediated dilation echocardiography Cardiac output Left ventricular stroke volume Isovolumic relaxation time deceleration time Pulse wave velocity High-resolution ultrasound nuclear factor (erythroid-derived 2)-like 2 developed pressure Blood pressure knock out wild type Translational research guanylyl cyclase

Related Topics

Life Sciences Biochemistry, Genetics and Molecular Biology Ageing

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Left ventricular diastolic dysfunction in Nrf2 knock out mice is associated with cardiac hypertrophy, decreased expression of SERCA2a, and preserved endothelial function

Authors

Ralf Erkens, Christian M. Kramer, Wiebke Lückstädt, Christina Panknin, Lisann Krause, Mathias Weidenbach, Jennifer Dirzka, Thomas Krenz, Evanthia Mergia, Tatsiana Suvorava, Malte Kelm, Miriam M. Cortese-Krott,