The neuroprotective properties of the superoxide dismutase mimetic tempol correlate with its ability to reduce pathological glutamate release in a rodent model of stroke

Article ID	Journal	Published Year	Pages	File Type
8269809	Free Radical Biology and Medicine	2014	45 Pages	PDF

Keywords

TBARS xCT Cystine/glutamate antiporter SIN-1 DHR123 DHK DCPIB MCAO intracerebroventricular ICV aCSF DTNB TTC tPA DTT DPPH CX43 BHT MCA SDS 2,2-diphenyl-1-picrylhydrazyl 2,3,5-triphenyltetrazolium chloride 3-morpholinosydnonimine 5,5′-dithiobis-(2-nitrobenzoic acid)DMSO Antioxidants superoxide anion 18α-glycyrrhetinic acid glutamate release middle cerebral artery occlusion Oxidative stress minimal essential medium dihydrorhodamine 123 dihydrokainate dithiothreitol Dimethyl sulfoxide Free radicals SOD sodium dodecyl sulfate Superoxide dismutase Stroke middle cerebral artery tissue plasminogen activator VRAC MEM artificial cerebrospinal fluid Neuroprotection thiobarbituric acid-reactive substances butylated hydroxytoluene volume-regulated anion channel connexin 43

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The neuroprotective properties of the superoxide dismutase mimetic tempol correlate with its ability to reduce pathological glutamate release in a rodent model of stroke

Authors

Preeti Dohare, MarÃa C. Hyzinski-GarcÃa, Aarshi Vipani, Nicole H. Bowens, Julia W. Nalwalk, Paul J. Feustel, Richard W. Keller Jr., David Jourd'heuil, Alexander A. Mongin,