Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
8272663 | Journal of the Neurological Sciences | 2018 | 25 Pages |
Abstract
Our results suggest that the Necl2 mutation identified herein may be associated with the risk of developing NMOSD. Furthermore, mutated NECL2 may play a role in the pathogenesis of the disease, potentially through its roles in axonal regeneration and/or via neuron-glia interactions that are relevant to myelination.
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Authors
Yan Xu, Liang Li, Hai-tao Ren, Bin Yin, Jian-gang Yuan, Xiao-zhong Peng, Bo-qin Qiang, Li-ying Cui,