| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8272836 | Journal of the Neurological Sciences | 2018 | 4 Pages |
Abstract
- Cardiac failure could be initially involved in transthyretin (TTR)-familial amyloid polyneuropathy (FAP) with Glu61Lys mutation.
- Okayama in Japan may be one of an endemic focus of TTR Glu61Lys variant.
- Constructive genetic examination is required in order to distinguish late onset FAP from senile systemic amyloidosis.
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Authors
Yumiko Nakano, Koh Tadokoro, Yasuyuki Ohta, Kota Sato, Mami Takemoto, Nozomi Hishikawa, Toru Yamashita, Taro Yamashita, Yukio Ando, Koji Abe,