Article ID Journal Published Year Pages File Type
8275513 Journal of the Neurological Sciences 2015 8 Pages PDF
Abstract
In patients with CMT1A, DM exacerbated clinical and pathological manifestations including increased loss of myelinated fibers, abnormal axon-myelin interaction, and impaired nerve regeneration.
Related Topics
Life Sciences Biochemistry, Genetics and Molecular Biology Ageing
Authors
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