Article ID Journal Published Year Pages File Type
8277243 Journal of the Neurological Sciences 2014 6 Pages PDF
Abstract
Our meta-analysis suggested that SMN1 duplications are a genetic risk factor in SALS, while there was no modulator effect of the SMN2 gene. In addition, it was possible that SMN1 deletions in predisposition to SALS vary across different countries. More studies were required to warrant the findings of this study.
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Related Topics
Life Sciences Biochemistry, Genetics and Molecular Biology Ageing
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SMN1 duplications contribute to sporadic amyotrophic lateral sclerosis susceptibility: Evidence from a meta-analysis
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