GSNOR modulates hyperhomocysteinemia-induced T cell activation and atherosclerosis by switching Akt S-nitrosylation to phosphorylation

Article ID	Journal	Published Year	Pages	File Type
8286463	Redox Biology	2018	30 Pages	PDF

Keywords

GSNO N-acetyl-l-cysteine IFN-γ iNOS IL-2 NAC Hcy DTT S-nitrosoglutathione HHcy PBMC S-nitrosoglutathione reductase S-nitrosothiols GSNOR ROS Atherosclerosis Apoe apolipoprotein E Akt post-translational modification Oxidoreductase interferon γ interleukin 2 coronary artery disease dithiothreitol Peripheral blood mononuclear cell T cell inducible nitric oxide synthase CAD Nitric oxide homocysteine Hyperhomocysteinemia Reactive oxygen species

Related Topics

Life Sciences Biochemistry, Genetics and Molecular Biology Ageing

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GSNOR modulates hyperhomocysteinemia-induced T cell activation and atherosclerosis by switching Akt S-nitrosylation to phosphorylation

Authors

Jing Li, Yan Zhang, Yuying Zhang, Silin Lü, Yutong Miao, Juan Yang, Shenming Huang, Xiaolong Ma, Lulu Han, Jiacheng Deng, Fangfang Fan, Bo Liu, Yong Huo, Qingbo Xu, Chang Chen, Xian Wang, Juan Feng,