| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8451390 | Experimental Cell Research | 2018 | 6 Pages |
Abstract
The S38G mutation induced a loss-of-function of IKs due to decreasing of KCNE1 protein expression and defecting in KCNE1 protein membrane trafficking. Our findings suggested that KCNE1 may be one of the possible modulatory genes associated to ERS.
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Authors
Hao Yao, Cheng-Cheng Ji, Yun-Jiu Cheng, Xu-Miao Chen, Li-Juan Liu, Jun Fan, Su-Hua Wu,