| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8458071 | Pathology - Research and Practice | 2018 | 41 Pages |
Abstract
MiR-136-5p is overexpressed in lung adenocarcinoma and is involved in the molecular mechanism of lung adenocarcinoma via suppressing the expressions of downstream targets, especially claudin-18, sialophorin and syndecan 2 that participate in cell adhesion.
Keywords
ROCGEOSROCSyndecanSTRINGPPIreads per millionTCGASMDSPESPNRPMDEGsmiRNAsNLRPLRGEPIAEGFRNegr1AUCCAMsALKSENSearch Tool for the Retrieval of Interacting Genesstandard deviationThe cancer genome atlasIHCImmunohistochemistrySCLCBioinformaticsMolecular functionGene Expression Profiling Interactive AnalysisProtein ANalysis THrough Evolutionary RelationshipsProtein-protein interactionStandard mean differencecellular componentSensitivityKEGG یا Kyoto Encyclopedia of Genes and Genomes Kyoto Encyclopedia of Genes and GenomesDORDAVIDmicroRNAslung adenocarcinomaNon small cell lung cancerNSCLCSmall cell lung cancerVascular endothelial growth factor AMeta-analysisbiological processAnaplastic lymphoma kinaseLUADreceiver operating curvearea under curvemeancell adhesion moleculespositive likelihood rationegative likelihood ratiodiagnostic odds ratioNeurofascinGene ontologySpecificityPANTHERdatabase for annotation, visualization and integrated discoveryGene Expression OmnibusDifferentially expressed genesEpidermal growth factor receptor
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Authors
Tian-tian Li, Xiang Gao, Li Gao, Bin-liang Gan, Zu-cheng Xie, Jing-jing Zeng, Gang Chen,