| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8473551 | Journal of Molecular and Cellular Cardiology | 2018 | 33 Pages |
Abstract
These data identify a key mechanistic link between transient PKC activation and long-term Ca2+ handling defects through PKC-induced actin cytoskeletal rearrangement and resultant T-tubule damage.
Related Topics
Life Sciences
Biochemistry, Genetics and Molecular Biology
Cell Biology
Authors
Ang Guo, Rong Chen, Yihui Wang, Chun-Kai Huang, Biyi Chen, William Kutschke, Jiang Hong, Long-Sheng Song,
![First Page Preview: Transient activation of PKC results in long-lasting detrimental effects on systolic [Ca2+]i in cardiomyocytes by altering actin cytoskeletal dynamics and T-tubule integrity Transient activation of PKC results in long-lasting detrimental effects on systolic [Ca2+]i in cardiomyocytes by altering actin cytoskeletal dynamics and T-tubule integrity](/preview/png/8473551.png)