| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8473706 | Journal of Molecular and Cellular Cardiology | 2016 | 29 Pages |
Abstract
EndMT has been recognised as a possible pathological mechanism for MVP. For the first time, we report the involvement of OPG in cellular and molecular changes in MVP isolated cells. In addition, we detected elevated circulating OPG levels in MVP patients when compared to controls, which supports the hypothesis that OPG is involved in MVP development and progression.
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Authors
Paola Songia, Emanuela Branchetti, Alessandro Parolari, Veronika Myasoedova, Giovanni Ferrari, Francesco Alamanni, Elena Tremoli, Paolo Poggio,