| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8473720 | Journal of Molecular and Cellular Cardiology | 2016 | 28 Pages |
Abstract
Ca++ ALT is suppressed by hMSCs in a paracrine fashion due to activation of a PI3K-mediated nitroso-redox pathway. These findings demonstrate, for the first time, how stem cell therapy might be antiarrhythmic by suppressing cardiac alternans through paracrine action on Ca++ cycling.
Related Topics
Life Sciences
Biochemistry, Genetics and Molecular Biology
Cell Biology
Authors
Prasongchai Sattayaprasert, Drew M. Nassal, Xiaoping Wan, Isabelle Deschenes, Kenneth R. Laurita,