Article ID Journal Published Year Pages File Type
8473928 Journal of Molecular and Cellular Cardiology 2016 12 Pages PDF
Abstract
These data suggested a role of EndoII in protecting BASMCs apoptosis induced by H2O2, possibly by inhibiting the addressing of Bax to mitochondria. Targeting on EndoII may be a new strategy to treat apoptosis-associated diseases.
Related Topics
Life Sciences Biochemistry, Genetics and Molecular Biology Cell Biology
Authors
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