| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8474256 | Journal of Molecular and Cellular Cardiology | 2015 | 9 Pages |
Abstract
Conclusions. Our study suggests that the increased acetylation of CyPD following myocardial ischemia-reperfusion facilitates PTP opening and subsequent cell death. Therefore ischemic postconditioning might prevent lethal reperfusion injury through an increased SIRT3 activity and subsequent attenuation of CyPD acetylation at reperfusion.
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Authors
T. Bochaton, C. Crola-Da-Silva, B. Pillot, C. Villedieu, L. Ferreras, M.R. Alam, H. Thibault, M. Strina, A. Gharib, M. Ovize, D. Baetz,