Reciprocal interactions between mitral valve endothelial and interstitial cells reduce endothelial-to-mesenchymal transition and myofibroblastic activation

Article ID	Journal	Published Year	Pages	File Type
8474405	Journal of Molecular and Cellular Cardiology	2015	11 Pages	PDF

Keywords

eNOS VICS chondromodulin-1 VECs EndMT ECFCs EBM ECM TGFβ BM-MSCs FITC qPCR MMP α-SMA FBS BSA Endothelial to mesenchymal transition VE-cadherin bovine serum albumin Endothelial-to-mesenchymal transition α-smooth muscle actin Transforming growth factor β mitral valve conditioned media fetal bovine serum Bone marrow-derived mesenchymal stem cells Valve interstitial cells Endothelial cells Endothelial colony forming cells endothelial nitric oxide synthase fluorescein isothiocyanate Extracellular matrix matrix metalloproteinase Quantitative reverse transcriptase polymerase chain reaction

Related Topics

Life Sciences Biochemistry, Genetics and Molecular Biology Cell Biology

Reciprocal interactions between mitral valve endothelial and interstitial cells reduce endothelial-to-mesenchymal transition and myofibroblastic activation

Authors

Kayle Shapero, Jill Wylie-Sears, Robert A. Levine, John E. Jr., Joyce Bischoff,